Brain neuron-derived WDFY1 induces bone loss
https://www.nature.com/articles/s43587-025-01032-8
Published: January 5, 2026 00:00
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How long will we live? And how much of that time will comprise a healthy life? What is aging, and can we stop or even reverse the aging process? What is the connection between aging and disease? Can we predict the evolving trends in the aging of human populations and prepare our societies for what has been called the Silver Tsunami? These are some of the important questions that the broad field of aging research is trying to address and that together form one of the Grand Challenges of the twenty-first century. The mission of Nature Aging is to provide a unique multidisciplinary, unifying and highly visible publishing platform for the aging-research community. The journal is highly selective yet broad in its coverage, publishing research from across the entire spectrum of the field, ranging from the basic biology of aging to the impact of aging on society. The journal aims to foster interactions among different areas of this diverse field of research and to promote new and exciting ideas within and beyond the research community, to enable synergy and maximize scientific and societal impact.
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Brain neuron-derived WDFY1 induces bone loss
https://www.nature.com/articles/s43587-025-01032-8
Published: January 5, 2026 00:00
Shear stress governs hematopoietic stem cell fate to promote inflammation-induced aging
https://www.nature.com/articles/s43587-025-01039-1
Published: January 2, 2026 00:00
Linking single-cell multiomics with GWAS to reveal key regulators of disease risk
https://www.nature.com/articles/s43587-025-01047-1
Published: January 2, 2026 00:00
Squishing, squeezing and stretching age hematopoietic stem cells
https://www.nature.com/articles/s43587-025-01044-4
Published: January 2, 2026 00:00
Rejuvenating the aging gut by targeting senescence
https://www.nature.com/articles/s43587-025-01045-3
Published: January 2, 2026 00:00
Cancer-cell-secreted DDAH1 induces TGF-β1/Smad3 signaling pathway to promote fibrosis and aging in lung
https://www.nature.com/articles/s43587-025-01024-8
Published: December 24, 2025 00:00
Allosteric activation of a cell-type-specific GPR120 inhibits amyloid pathology of Alzheimer’s disease
https://www.nature.com/articles/s43587-025-01028-4
Published: December 19, 2025 00:00
Repurposing glucagon-like peptide-1 receptor agonists for the treatment of neurodegenerative disorders
https://www.nature.com/articles/s43587-025-01029-3
Published: December 19, 2025 00:00
CDK3 induces neuronal death and brain atrophy in Alzheimer’s disease
https://www.nature.com/articles/s43587-025-01026-6
Published: December 19, 2025 00:00
Integrating polygenic signals and single-cell multiomics identifies cell-type-specific regulomes critical for immune- and aging-related diseases
https://www.nature.com/articles/s43587-025-01027-5
Published: December 17, 2025 00:00
Author Correction: Aging by the clock and yet without a program
https://www.nature.com/articles/s43587-025-01050-6
Published: December 15, 2025 00:00
GDF3 promotes adipose tissue macrophage-mediated inflammation via altered chromatin accessibility during aging
https://www.nature.com/articles/s43587-025-01034-6
Published: December 15, 2025 00:00
An assessment of intrinsic capacity among older Indian adults from the Longitudinal Ageing Study in India
https://www.nature.com/articles/s43587-025-01013-x
Published: December 12, 2025 00:00
Reframing biological age as risk-equivalent age
https://www.nature.com/articles/s43587-025-01038-2
Published: December 11, 2025 00:00
Microbiota safeguards ovarian reserve and extend reproductive lifespan
https://www.nature.com/articles/s43587-025-01042-6
Published: December 5, 2025 00:00