Cell populations in human breast cancers are molecularly and biologically distinct with age
https://www.nature.com/articles/s43587-025-00984-1
Published: November 4, 2025 00:00
@feeds.nature.com.nataging.rss.current@rss-parrot.net
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How long will we live? And how much of that time will comprise a healthy life? What is aging, and can we stop or even reverse the aging process? What is the connection between aging and disease? Can we predict the evolving trends in the aging of human populations and prepare our societies for what has been called the Silver Tsunami? These are some of the important questions that the broad field of aging research is trying to address and that together form one of the Grand Challenges of the twenty-first century. The mission of Nature Aging is to provide a unique multidisciplinary, unifying and highly visible publishing platform for the aging-research community. The journal is highly selective yet broad in its coverage, publishing research from across the entire spectrum of the field, ranging from the basic biology of aging to the impact of aging on society. The journal aims to foster interactions among different areas of this diverse field of research and to promote new and exciting ideas within and beyond the research community, to enable synergy and maximize scientific and societal impact.
Site URL: feeds.nature.com/nataging/rss/current
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Cell populations in human breast cancers are molecularly and biologically distinct with age
https://www.nature.com/articles/s43587-025-00984-1
Published: November 4, 2025 00:00
A unified framework for systematic curation and evaluation of aging biomarkers
https://www.nature.com/articles/s43587-025-00987-y
Published: November 4, 2025 00:00
A focus shift from sarcopenia to muscle health in the Asian Working Group for Sarcopenia 2025 Consensus Update
https://www.nature.com/articles/s43587-025-01004-y
Published: November 4, 2025 00:00
Aging represses oncogenic KRAS-driven lung tumorigenesis and alters tumor suppression
https://www.nature.com/articles/s43587-025-00986-z
Published: November 4, 2025 00:00
Hospital-treated infectious diseases and the risk of epilepsy in older age
https://www.nature.com/articles/s43587-025-01005-x
Published: November 4, 2025 00:00
A synthetic oocyte aging method for uncovering the molecular origins of egg aneuploidy
https://www.nature.com/articles/s43587-025-01010-0
Published: November 4, 2025 00:00
A versatile cohesion manipulation system probes female reproductive age-related egg aneuploidy
https://www.nature.com/articles/s43587-025-00997-w
Published: November 3, 2025 00:00
Effect of the mitophagy inducer urolithin A on age-related immune decline: a randomized, placebo-controlled trial
https://www.nature.com/articles/s43587-025-00996-x
Published: October 31, 2025 00:00
Adapting health, economic and social policies to address population aging in China
https://www.nature.com/articles/s43587-025-00999-8
Published: October 31, 2025 00:00
Loss of MFE-2 impairs microglial lipid homeostasis and drives neuroinflammation in Alzheimer’s pathogenesis
https://www.nature.com/articles/s43587-025-00976-1
Published: October 29, 2025 00:00
Aging as a glitch in the matrix
https://www.nature.com/articles/s43587-025-01009-7
Published: October 29, 2025 00:00
Epigenetic dysregulation in aged muscle stem cells drives mesenchymal progenitor expansion via IL-6 and Spp1 signaling
https://www.nature.com/articles/s43587-025-01002-0
Published: October 29, 2025 00:00
Fat modifications protect joints from degrading
https://www.nature.com/articles/s43587-025-01003-z
Published: October 24, 2025 00:00
Single-cell analysis of human thymus and peripheral blood unveils the dynamics of T cell development and aging
https://www.nature.com/articles/s43587-025-00990-3
Published: October 24, 2025 00:00
Author Correction: Mitochondria-associated condensates maintain mitochondrial homeostasis and promote lifespan
https://www.nature.com/articles/s43587-025-01015-9
Published: October 22, 2025 00:00